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Is troponin-I elevated during prolonged status epilepticus associated with sympathetic outflow?

Introduction: To determine the clinical significance of elevation of Troponin-I [cTn-I] during prolonged status epilepticus [pSE] SE is known to be accompanied by an increase in sympathetic outflow. Elevation of cTn-I has been linked to myocardial stress.

What are the diagnostic criteria for status epilepticus (SE)?

MGH STATUS EPILEPTICUS TREATMENT PROTOCOL DIAGNOSIS OF STATUS EPILEPTICUS: Generalized convulsive status epilepticus Continuous convulsive seizure activity lasting > 5 mins OR, ≥ 2 convulsive seizures without full return to baseline between seizures

What causes elevated uric acid and troponin levels?

In cases involving postictal, acute renal failure without rhabdomyolysis, the elevated uric acid could be the culprit. Troponin elevations occasionally point towards myocardial infarction, stress cardiomyopathy or other grave cardiac complications, but are benign in most cases.

What blood tests are used to diagnose generalized tonic-clonic seizures (GTCS)?

Elevations in the creatine kinase (CK) levels are common after generalized tonic-clonic seizures (GTCS) and display high specificity and moderate sensitivity. Metabolic markers such as ammonia and lactate may have diagnostic potential for postictal blood tests.


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